Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation yanayi ne na fata wanda ke haifar da ƙara launi a fata bayan kumburi. Postinflammatory hyperpigmentation na iya faruwa sakamakon tsawaita fitowar rana, kumburi, ko wasu raunin fata, har ma da kuraje. Mutane masu launin fata masu duhu yawanci sukan fi fuskantar hyperpigmentation, musamman idan sun yi tsawaita fitowar rana.
☆ AI Dermatology — Free Service
A cikin sakamakon Stiftung Warentest na 2022 daga Jamus, gamsuwar mabukaci tare da ModelDerm ya ɗan yi ƙasa kaɗan fiye da biyan shawarwarin telemedicine.
A cikin sakamakon Stiftung Warentest na 2022 daga Jamus, gamsuwar mabukaci tare da ModelDerm ya ɗan yi ƙasa kaɗan fiye da biyan shawarwarin telemedicine.
relevance score : -100.0%
ReferencesPostinflammatory hyperpigmentation (PIH) wannan shine matsalar fata da ake yawan samu bayan kumburin fata ko rauni. Yakan ɗaukar lokaci mai tsawo kuma ya fi muni a tsakanin mutane masu launin fata (Fitzpatrick skin types III–VI). Ko da yake sau da yawa yana warkewa da kansa, wannan na iya ɗaukar ɗan lokaci; don haka a lokuta da dama ana buƙatar magani na dogon lokaci. Haɗa magunguna daban‑daban yana aiki mafi kyau.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation shi ne yanayin da ke faruwa bayan kumburin fata. Yana fi shafar mutanen da ke da launin fata mai duhu da kuma waɗanda ke fama da kumburi akai‑akai. Nazarin ya nuna cewa batutuwa kamar postinflammatory hyperpigmentation suna daga cikin manyan dalilan da ke sa masu launin fata su nemi kulawar fata. Magani na farko yana da muhimmanci don magance postinflammatory hyperpigmentation, kuma yawanci yana farawa da sarrafa kumburin farko. Layin farko na jiyya yawanci ya haɗa da amfani da magunguna masu haske waɗanda ke rage ƙalar fata tare da kariyar hasken rana. Waɗannan wakilai, kamar hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts, suna iya rage yawan melanin yadda ya kamata. Bugu da ƙari, retinoids, mequinol, ascorbic acid, niacinamide, N‑acetyl glucosamine, da soy ana amfani da su azaman wakilai masu gyara launi, tare da sabbin hanyoyin jiyya da ke fitowa. Duk da yake jiyya na gida galibi suna da tasiri ga matakin hyperpigmentation, hanyoyin (laser, chemical peel) na iya zama dole a lokuta masu tsanani. Yana da muhimmanci a yi amfani da takatsantsan tare da waɗannan jiyya don guje wa ƙara tsananta postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
